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Although retrieval of memories requires, obligatorily, that the information has been acquired at some point, its mere acquisition does not make this information a memory per se.


The information only becomes a memory when information is learned, stored 11 and subsequently retrieved. Information acquisition can occur in a variety of ways, albeit through empirical experiences, stimuli captured by our sensory and somesthetic systems, or even through situations created in our own thoughts. The process of information selection is important to enable systems involved in memory to refine, amongst all the complexity of information it receives, which items are most relevant based on the focus given to the information, emotions involved with it, among other as yet unknown systems of refinement.

According to Cassini, 16 there is evidence that with time, hippocampal activity decreases in response to the retrieval of distant memories. On the other hand, an increase in cortical activity takes place.

This suggests that memory progressively, dynamically and in a plastic manner, becomes independent of the hippocampus and more dependent on cortical regions, a process called Systemic Consolidation or Reorganization of Memory.


CHOI, S. Antinociceptive profiles of aspirin and acetaminophen in formalin, substance P and glutamate pain models. Brain Research, v.

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Department of Health and Human Services. The Journal of Biological Chemistry, v. Pharmacology of cannabinoid receptors. Annual Review of Pharmacology and Toxicology, v. Pediatrics, v. Capsaicin in the periaqueductal gray induces analgesia via metabo-tropic glutamate receptor-mediated endocannabinoid retrograde disinhibition. British Journal of Pharmacology, v. Journal of Pharmacology and Experimental Therapeutics, v.

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TRPV1 in brain is involved in acetaminophen-induced antinociception. PLoS one, v. Mechanism of acetaminophen inhibition of cyclooxygenase isoforms. Archives of Biochemistry and Biophysics, v.

Os pesquisadores recrutaram 8. Topol e cols. Assim, lumiracoxib foi considerado, pelos pesquisadores do TARGET, como sendo o tratamento ideal para os pacientes com osteoartrite. Ray e cols.

Juni e cols. Em , Solomon e cols. Liberados em setembro de , causaram a pronta retirada do rofecoxib do mercado por parte da empresa que o comercializava.

Um total de 1.

O risco relativo obtido foi de 1. Pesquisadores do APC discutiram resultados de outros estudos envolvendo celecoxib. Essa conduta do FDA levou a empresa produtora do rofecoxib a indicar que, em breve, deve retornar a comercializar o produto. Complications of the COX-2 inhibitors parecoxib and valdecoxib after cardiac surgery.

N Engl J Med ; The coxibs, selective inhibitors of cycloxygenase Lessons from the withdrawal of rofecoxib.

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Br Med J ; Risk of cardiovascular events associated with selective COX-2 inhibitors. JAMA ; Gastrointestinal toxicity with celecoxib vs nonsteroidal anti-inflammatory drugs for osteoarthritis and rheumatoid arthritis: the CLASS study: a randomized controlled trial. Rio de Janeiro: Guanabara Koogan, FitzGerald GA. Cardiovascular pharmacology of nonselective nonsteroidal anti-inflammatory drugs and coxibs: clinical considerations.

Am J Cardiol ; 26DD. Comparison of upper gastrointestinal toxicity of rofecoxib and naproxen in patients with rheumatoid arthritis. Cyclooxygenase-2 and its regulation in inflammation.

Mediators Inflamm ; 5: Pharmacological and biochemical demonstration of the role of cyclooxygenase-2 in inflammation and pain.

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